2 edition of Factors affecting uterine blood flow in the rat found in the catalog.
Factors affecting uterine blood flow in the rat
Sabah S M. Phaily
Written in English
Ph.D. thesis. Typescript.
|The Physical Object|
|Number of Pages||187|
Uterine and placental blood flow during pregnancy. Early (–) human studies by Assali et al (3, 4) and Metcalfe utilizing the diffusion equilibrium principle (most often nitrous oxide, N 2 O) or electro-magnetic flow probes placed directly on the uterine artery reported that total uteroplacental blood flow (UPBF) increases from a baseline value of 20–50 ml/min to – ml/min in. Effects of Different Intensities of Acupuncture Stimuli on the Rat's Uterine Microvascular Blood Flow in Each Period. Table 1 shows the effect of acupuncture with two different stimuli on uterus blood flow of female rats at different period. which implies that hand manipulation is one of the important factors affecting the curative.
Any factor that causes cardiac output to increase, by elevating heart rate or stroke volume or both, will elevate blood pressure and promote blood flow. These factors include sympathetic stimulation, the catecholamines epinephrine and norepinephrine, thyroid hormones, and increased calcium ion levels. Delayed pregnancy in the rat reduced fertility by 46%, reduced litter size by 36%, caused fetal growth restriction, increased placental weight, and increased maternal systolic blood pressure (by 16 mm Hg). Uterine arteries from aged dams displayed reduced constriction to phenylephrine (young: ± mN/mm versus aged: ± mN/mm, P=0.
Absolute flow to the ovaries increased nearly 5‐fold from the NP state ( ± ) to day 20 of pregnancy ( ± ). Interlitter differences in ovarian blood flow during midgestation were found to be a result of differences in litter size and distribution of embryo/fetuses between the two uterine horns. Mean arterial blood pressure, heart rate, uterine artery blood flow (UtBF), uterine artery conductance (UtC), and the relative changes in uterine artery blood flow and conductance at each stage of gestation in rabbits measured at rest (time 0) and during graded exercise (7% grade) on a treadmill at 7, 10, and 13 m/min and at maximal (max.
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Frederick C. Battaglia, in Fetal and Neonatal Physiology (Third Edition), Uterine Blood Flow. Measurements of uterine blood flow in human pregnancies were first reported in the s. 20–22 These studies reported estimates of uterine blood flow of approximately mL/minute at term in normal pregnancies.
Meschia 12 pointed out that these estimates were likely to be too low, based on. Factors Affecting the Determination of Uterine Blood Flow in Vivo By WILLIAM E.
HUCKABEE, M.D., AND DONALD H. BAERON, PH.D. BECAUSE no other organ of the mam-malian body has so complex an internal structure as the pregnant uterus, measure-ment of blood flow to the uterus presents some unique features that require analysis.
The. Minimal change in uterine blood flow (sheep studies [Fresno L et al. Vet J 94, ]) Ketamine. No change in uterine blood flow using mg/kg (sheep studies [Craft JB Jr. et al. Am J Obs Gyn]) Volatile Anesthetics. Isoflurane preserves uterine blood flow despite the decrease in MAP, most likely due to uterine vasodilation.
The book Miraculous Pivot-Ni Shun Fei Shou has mentioned, Effects of Different Intensities of Acupuncture Stimuli on the Rat's Uterine Microvascular Blood Flow in Each Period.
which implies that hand manipulation is one of the important factors affecting the curative effect. Our finding highlights using thick needles and deep insertion Cited by: 4.
Factors Affecting Uterine Blood Flow in the Rat. Author: Phaily, S. ISNI: Awarding Body: University of Bradford Current Institution: University of Bradford Date of Award: Availability of Full Text: Full text unavailable from EThOS.
Keywords: axon reflex, cerebral blood flow, muscle blood flow, somato-autonomic reflex, uterine blood flow Introduction Somatic afferent stimulation to the skin, muscle and joints has been found to produce reflex responses in visceral function via autonomic efferent nerves in animals anesthetized to eliminate emotional factors (1).
In singleton pregnancies at term, blood flow to the uterus is approximately to mL/minute. The increased blood flow is directly related to the increasing uterine size and results from a combination of the increased cardiac output of pregnancy and uterine demand. SUMMARY. While evidence is accumulating that prostaglandin F 2α (PGF 2α) is the uterine luteolytic factor in several sub-primate species, factors controlling the release of PGF 2α from the uterus are not fully documented.
The present study utilizes two models consisting of either the in situ or the autotransplanted uterus of the sheep. Four factors affecting the release of PGF 2α from the.
Uterine fibroids, known medically as leiomyomas, are benign growths of uterine muscle. Genetics, race, hormones and previous pregnancies can all have a role in creating fibroids.
A normal size of the uterine body ( × mm: long axis × anteroposterior diameter) and endometrial thickness (yellow arrow) were found, with sufficient uterine blood flow. (B) Findings in an animal with warm ischemia for 8 h, in which menstruation.
Fig. Changes in uterine arteriovenous oxygen extraction—(A-V)0 2 — in response to reductions in uterine blood flow during the second half of ovine gestation. Since (A-V)02 can be doubled, uterine 02 delivery will be maintained at homeostatic levels until uterine blood flow is reduced more than 50%.
The clear, stippled, and lined areas indicate hypothetical degrees of fetal reserve when. Effects of late-gestation (E–) hypoxia on fetal growth and uterine artery blood flow. A) E fetal weight was reduced, B) placental weight was increased, and therefore C) the ratio of fetal to placental weight was lower indicating a selective reduction in fetal growth; n = 10 normoxic, 10 hypoxic dams.D) Uterine artery volumetric blood flow was increased with late-gestation hypoxia.
Increased E2:P4 is known to increase mean blood flow to many vascular beds, but especially the uterus (2, ). Local, exogenous treatment with E2 can also increase blood flow to the uterine vascular bed (9, 12, 16).
Since blood pressure does not change significantly over the ovarian cycle, these increases in blood flow are enabled by a drop.
An analysis of the contribution of each artery (ovarian and uterine) to the total blood flow summed over all genitalia (ovaries, oviducts and uterine horns) indicated that on both Days 7 and The temporary interruption of uterine blood flow to one horn of the rat uterus leads to reduced fetal growth in not only the temporarily ischemic horn but also the contralateral horn.
This suggests that the reduction of fetal growth is caused by not only the temporary interruption of blood flow to the uteroplacental unit(s) but also circulating.
Dynamic Control of Placental Blood Flow. A schematic representation of those factors affecting placental and non-placental blood flow in the uterus is shown in Fig. At term gestation in women, the pro portion of total UBF perfusing the placenta probably exceeds.
The uterine vein refers to a group of blood vessels found near the genitalia on the female body. These veins are considered part of a network of blood vessels called the uterine. Any increase in uterine vascular resistance will decrease uterine perfusion, as is seen with uterine contractions.
Of all factors ensuring the overall success of the EXIT procedure, minimal uterine vascular resistance is the most important because decreases in uterine blood flow will cause fetal hypoxemia, acidosis, and, potentially, fetal demise.
Inadequate placental blood flow impairs fetal growth and development and in turn may ‘programme’ the fetus to develop disorders in adulthood as evidenced by animal studies in which uterine blood flow has been experimentally reduced (Simmons et al.
In support of a role for uterine artery dysfunction in programming we. Many factors can influence a baby’s growth during pregnancy, including: A problem with the placenta, which causes decreased blood flow to the baby; Birth defects or genetic abnormalities; Health problems in the mom-to-be, such as certain viral infections, blood pressure problems, cardiovascular disease, autoimmune disease, advanced diabetes.
It is well known that uterine arteries may play a major role in regulating uteroplacental blood flow. Reports indicate that the weight of these blood vessels increases 4-fold during pregnancy to accommodate the large increase in uterine blood flow. Cardiac output also increases during pregnancy, but its redistribution is not uniform.Reduced uterine perfusion pressure during pregnancy in the rat is associated with increases in arterial pressure and changes in renal nitric oxide.
Hypertension. ; – Crossref Medline Google Scholar; 13 Crews JK, Herrington JN, Granger JP, Khalil RA. Decreased endothelium-dependent vascular relaxation during reduction of.Yoshimasa Kamei's 80 research works with 1, citations and 4, reads, including: Qualitative investigation of the factors that generate ambivalent feelings in women who give birth after.